Angiotensin Receptor-Neprilysin Inhibition in Acute Myocardial Infarction


Por: Pfeffer, MA, Claggett, B, Lewis, EF, Granger, CB, Kober, L, Maggioni, AP, Mann, DL, McMurray, JJV, Rouleau, JL, Solomon, SD, Steg, PG, Berwanger, O, Cikes, M, De Pasquale, CG, East, C, Fernandez, A, Jering, K, Landmesser, U, Mehran, R, Merkely, B, Mody, FV, Petrie, MC, Petrov, I, Schou, M, Senni, M, Sim, D, van der Meer, P, Lefkowitz, M, Zhou, Y, Gong, J, Braunwald, E, Sionis A., PARADISE-MI Invest & Comm

Publicada: 11 nov 2021
Resumen:
Sacubitril-Valsartan in Acute Myocardial Infarction In a randomized trial, 5661 patients with acute myocardial infarction and a reduced left ventricular ejection fraction, pulmonary congestion, or both were assigned to receive either sacubitril-valsartan or ramipril. At a median of 22 months, there was no significant difference between the two groups in the incidence of death from cardiovascular causes or incident heart failure. Background In patients with symptomatic heart failure, sacubitril-valsartan has been found to reduce the risk of hospitalization and death from cardiovascular causes more effectively than an angiotensin-converting-enzyme inhibitor. Trials comparing the effects of these drugs in patients with acute myocardial infarction have been lacking. Methods We randomly assigned patients with myocardial infarction complicated by a reduced left ventricular ejection fraction, pulmonary congestion, or both to receive either sacubitril-valsartan (97 mg of sacubitril and 103 mg of valsartan twice daily) or ramipril (5 mg twice daily) in addition to recommended therapy. The primary outcome was death from cardiovascular causes or incident heart failure (outpatient symptomatic heart failure or heart failure leading to hospitalization), whichever occurred first. Results A total of 5661 patients underwent randomization; 2830 were assigned to receive sacubitril-valsartan and 2831 to receive ramipril. Over a median of 22 months, a primary-outcome event occurred in 338 patients (11.9%) in the sacubitril-valsartan group and in 373 patients (13.2%) in the ramipril group (hazard ratio, 0.90; 95% confidence interval [CI], 0.78 to 1.04; P=0.17). Death from cardiovascular causes or hospitalization for heart failure occurred in 308 patients (10.9%) in the sacubitril-valsartan group and in 335 patients (11.8%) in the ramipril group (hazard ratio, 0.91; 95% CI, 0.78 to 1.07); death from cardiovascular causes in 168 (5.9%) and 191 (6.7%), respectively (hazard ratio, 0.87; 95% CI, 0.71 to 1.08); and death from any cause in 213 (7.5%) and 242 (8.5%), respectively (hazard ratio, 0.88; 95% CI, 0.73 to 1.05). Treatment was discontinued because of an adverse event in 357 patients (12.6%) in the sacubitril-valsartan group and 379 patients (13.4%) in the ramipril group. Conclusions Sacubitril-valsartan was not associated with a significantly lower incidence of death from cardiovascular causes or incident heart failure than ramipril among patients with acute myocardial infarction. (Funded by Novartis; PARADISE-MI ClinicalTrials.gov number, .)

Filiaciones:
Pfeffer, MA:
 Brigham & Womens Hosp, Cardiovasc Div, 75 Francis St, Boston, MA 02115 USA

Claggett, B:
 Brigham & Womens Hosp, Cardiovasc Div, 75 Francis St, Boston, MA 02115 USA

Lewis, EF:
 Stanford Univ, Sch Med, Div Cardiovasc Med, Palo Alto, CA 94304 USA

Granger, CB:
 Duke Univ, Med Ctr, Durham, NC USA

Kober, L:
 Univ Copenhagen, Rigshosp, Blegdamsvej, Copenhagen, Denmark

Maggioni, AP:
 Natl Assoc Hosp Cardiologists Res Ctr, Florence, Italy

Mann, DL:
 Washington Univ, Sch Med, St Louis, MO USA

McMurray, JJV:
 Univ Glasgow, British Heart Fdn Cardiovasc Res Ctr, Glasgow, Lanark, Scotland

Rouleau, JL:
 Univ Montreal, Montreal Heart Inst, Montreal, PQ, Canada

Solomon, SD:
 Brigham & Womens Hosp, Cardiovasc Div, 75 Francis St, Boston, MA 02115 USA

Steg, PG:
 Univ Paris, AP HP, French Alliance Cardiovasc Trials, Paris, France

 INSERM, Unite 1148, Paris, France

Berwanger, O:
 Hosp Israelita Albert Einstein, Acad Res Org, Sao Paulo, Brazil

Cikes, M:
 Univ Zagreb, Sch Med, Univ Hosp Ctr Zagreb, Dept Cardiovasc Dis, Zagreb, Croatia

De Pasquale, CG:
 Flinders Med Ctr, Dept Cardiovasc Med, Adelaide, SA, Australia

East, C:
 Baylor Scott & White Heart & Vasc Hosp, Baylor Soltero CV Res Ctr, Dallas, TX USA

Fernandez, A:
 Sanatorio Modelo Quilmes, Serv Cardiol, Quilmes, Argentina

Jering, K:
 Brigham & Womens Hosp, Cardiovasc Div, 75 Francis St, Boston, MA 02115 USA

Landmesser, U:
 Charite Univ Med Berlin, Berlin Inst Hlth, German Ctr Cardiovasc Res Partner Site Berlin, Dept Cardiol, Berlin, Germany

Mehran, R:
 Icahn Sch Med Mt Sinai, Zena & Michael A Wiener Cardiovasc Inst, New York, NY 10029 USA

Merkely, B:
 Semmelweis Univ, Heart & Vasc Ctr, Budapest, Hungary

Mody, FV:
 Univ Calif Los Angeles, Dept Vet Affairs Greater Los Angeles, Heart Failure & Prevent Cardiol Programs, Los Angeles, CA USA

Petrie, MC:
 Univ Glasgow, British Heart Fdn Cardiovasc Res Ctr, Glasgow, Lanark, Scotland

Petrov, I:
 Acibadem City Clin Cardiovasc Ctr, Sofia, Bulgaria

Schou, M:
 Herlev Gentofte Univ Hosp, Dept Cardiol, Copenhagen, Denmark

Senni, M:
 Hosp Papa Giovanni XXIII, Cardiovasc Dept, Bergamo, Italy

Sim, D:
 Natl Heart Ctr Singapore, Singapore, Singapore

van der Meer, P:
 Univ Groningen, Univ Med Ctr Groningen, Dept Cardiol, Groningen, Netherlands

Lefkowitz, M:
 Novartis, E Hanover, NJ USA

Zhou, Y:
 Novartis, E Hanover, NJ USA

Gong, J:
 Novartis, E Hanover, NJ USA

Braunwald, E:
 Brigham & Womens Hosp, Cardiovasc Div, 75 Francis St, Boston, MA 02115 USA

 Brigham & Womens Hosp, Cardiovasc Div, Thrombolysis Myocardial Infarct Study Grp, 75 Francis St, Boston, MA 02115 USA

Sionis A.:
 Institut d’Investigació Biomèdica Sant Pau (IIB SANT PAU), Sant Quintí 77-79, 08041 Barcelona, Spain
ISSN: 00284793
Editorial
MASSACHUSETTS MEDICAL SOC, WALTHAM WOODS CENTER, 860 WINTER ST,, WALTHAM, MA 02451-1413 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 385 Número: 20
Páginas: 1845-1855
WOS Id: 000724699600011
ID de PubMed: 34758252
imagen Green Published

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