Pittsburgh compound B imaging and cerebrospinal fluid amyloid-beta in a multicentre European memory clinic study


Por: Leuzy, A, Chiotis, K, Hasselbalch, SG, Rinne, JO, de Mendonca, A, Otto, M, Lleo, A, Castelo-Branco, M, Santana, I, Johansson, J, Anderl-Straub, S, von Arnim, CAF, Beer, A, Blesa, R, Fortea, J, Herukka, SK, Portelius, E, Pannee, J, Zetterberg, H, Blennow, K, Nordberg, A

Publicada: 1 sep 2016
Resumen:
The aim of this study was to assess the agreement between data on cerebral amyloidosis, derived using Pittsburgh compound B positron emission tomography and (i) multi-laboratory INNOTEST enzyme linked immunosorbent assay derived cerebrospinal fluid concentrations of amyloid-beta(42); (ii) centrally measured cerebrospinal fluid amyloid-beta(42) using a Meso Scale Discovery enzyme linked immunosorbent assay; and (iii) cerebrospinal fluid amyloid-beta(42) centrally measured using an antibody-independent mass spectrometry-based reference method. Moreover, we examined the hypothesis that discordance between amyloid biomarker measurements may be due to interindividual differences in total amyloid-beta production, by using the ratio of amyloid-beta(42) to amyloid-beta(40). Our study population consisted of 243 subjects from seven centres belonging to the Biomarkers for Alzheimer's and Parkinson's Disease Initiative, and included subjects with normal cognition and patients with mild cognitive impairment, Alzheimer's disease dementia, frontotemporal dementia, and vascular dementia. All had Pittsburgh compound B positron emission tomography data, cerebrospinal fluid INNOTEST amyloid-beta(42) values, and cerebrospinal fluid samples available for reanalysis. Cerebrospinal fluid samples were reanalysed (amyloid-beta(42) and amyloid-beta(40)) using Meso Scale Discovery electrochemiluminescence enzyme linked immunosorbent assay technology, and a novel, antibody-independent, mass spectrometry reference method. Pittsburgh compound B standardized uptake value ratio results were scaled using the Centiloid method. Concordance between Meso Scale Discovery/mass spectrometry reference measurement procedure findings and Pittsburgh compound B was high in subjects with mild cognitive impairment and Alzheimer's disease, while more variable results were observed for cognitively normal and non-Alzheimer's disease groups. Agreement between Pittsburgh compound B classification and Meso Scale Discovery/mass spectrometry reference measurement procedure findings was further improved when using amyloid-beta(42/40). Agreement between Pittsburgh compound B visual ratings and Centiloids was near complete. Despite improved agreement between Pittsburgh compound B and centrally analysed cerebrospinal fluid, a minority of subjects showed discordant findings. While future studies are needed, our results suggest that amyloid biomarker results may not be interchangeable in some individuals.

Filiaciones:
Leuzy, A:
 Karolinska Inst, Div Translat Alzheimer Neurobiol, Ctr Alzheimer Res, Dept Neurobiol Care Sci & Soc, Stockholm, Sweden

Chiotis, K:
 Karolinska Inst, Div Translat Alzheimer Neurobiol, Ctr Alzheimer Res, Dept Neurobiol Care Sci & Soc, Stockholm, Sweden

Hasselbalch, SG:
 Rigshosp, Copenhagen Univ Hosp, Danish Dementia Res Ctr, Copenhagen, Denmark

Rinne, JO:
 Univ Turku, Turku Univ Hosp, Div Clin Neurosci, Turku, Finland

 Univ Turku, Turku PET Ctr, Turku, Finland

de Mendonca, A:
 Univ Lisbon, Fac Med, Dept Neurol, Lisbon, Portugal

 Univ Lisbon, Fac Med, Lab Neurosci, Lisbon, Portugal

Otto, M:
 Ulm Univ Hosp, Dept Neurol, Ulm, Germany

Lleo, A:
 Hosp Santa Creu & Sant Pau, Inst Invest Biomed, Dept Neurol, Barcelona, Spain

 Ctr Invest Biomed Red Enfermedades Neurodegenerat, Barcelona, Spain

Castelo-Branco, M:
 Brain Imaging Network Portugal, Inst Nucl Sci Appl Hlth ICNAS, Coimbra, Portugal

 Univ Coimbra, Inst Biomed Imaging & Life Sci IBILI, Coimbra, Portugal

 Univ Coimbra, Fac Med, Coimbra, Portugal

Santana, I:
 Coimbra Univ Hosp, Dept Neurol, Coimbra, Portugal

 Univ Coimbra, Fac Med, Ctr Neurosci & Cell Biol CNC, Coimbra, Portugal

Johansson, J:
 Univ Turku, Turku PET Ctr, Turku, Finland

Anderl-Straub, S:
 Ulm Univ Hosp, Dept Neurol, Ulm, Germany

von Arnim, CAF:
 Ulm Univ Hosp, Dept Neurol, Ulm, Germany

Beer, A:
 Ulm Univ Hosp, Dept Nucl Med, Ulm, Germany

Blesa, R:
 Hosp Santa Creu & Sant Pau, Inst Invest Biomed, Dept Neurol, Barcelona, Spain

 Ctr Invest Biomed Red Enfermedades Neurodegenerat, Barcelona, Spain

Fortea, J:
 Hosp Santa Creu & Sant Pau, Inst Invest Biomed, Dept Neurol, Barcelona, Spain

 Ctr Invest Biomed Red Enfermedades Neurodegenerat, Barcelona, Spain

Herukka, SK:
 Univ Eastern Finland, Dept Neurol, Kuopio, Finland

 Kuopio Univ Hosp, Kuopio, Finland

Portelius, E:
 Univ Gothenburg, Sahlgrenska Acad, Inst Neurosci & Physiol, Dept Psychiat & Neurochem, Molndal, Sweden

Pannee, J:
 Univ Gothenburg, Sahlgrenska Acad, Inst Neurosci & Physiol, Dept Psychiat & Neurochem, Molndal, Sweden

Zetterberg, H:
 Univ Gothenburg, Sahlgrenska Acad, Inst Neurosci & Physiol, Dept Psychiat & Neurochem, Molndal, Sweden

 UCL Inst Neurol, Dept Mol Neurosci, London, England

Blennow, K:
 Univ Gothenburg, Sahlgrenska Acad, Inst Neurosci & Physiol, Dept Psychiat & Neurochem, Molndal, Sweden

Nordberg, A:
 Karolinska Inst, Div Translat Alzheimer Neurobiol, Ctr Alzheimer Res, Dept Neurobiol Care Sci & Soc, Stockholm, Sweden

 Karolinska Univ Hosp Huddinge, Dept Geriatr Med, Stockholm, Sweden
ISSN: 00068950





BRAIN
Editorial
OXFORD UNIV PRESS, GREAT CLARENDON ST, OXFORD OX2 6DP, ENGLAND, Reino Unido
Tipo de documento: Article
Volumen: 139 Número:
Páginas: 2540-2553
WOS Id: 000383719500026
ID de PubMed: 27401520
imagen Green Published, Hybrid Gold

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