The no-reflow phenomenon in the young and in the elderly


Por: Cenko, E, Ricci, B, Kedev, S, Kalpak, O, Calmac, L, Vasiljevic, Z, Knezevic, B, Dilic, M, Milicic, D, Manfrini, O, Koller, A, Dorobantu, M, Badimon, L, Bugiardini, R

Publicada: 1 nov 2016
Resumen:
Background: The objectives of this study were to evaluate the incidence of no-reflow as independent predictor of adverse events and to assess whether baseline pre-procedural treatment options may affect clinical outcomes. Methods: Data were derived from the ISACS-TC registry (NCT01218776) from October 2010 to January 2015. No-reflow was defined as post-PCI TIMI flow grades 0-1, in the absence of post-procedural significant (>= 25%) residual stenosis, abrupt vessel closure, dissection, perforation, thrombus of the original target lesion, or epicardial spasm. The outcome measure was in-hospital mortality. Results: No-reflow was identified in 128 of 5997 patients who have undergone PCI (2.1%). On multivariate analysis, patients with no-reflow were more likely to be older (OR: 1.20, 95% CI: 1.01-1.44), to have a history of hypercho-lesterolemia (OR: 1.95, 95% CI: 1.31-2.91) and to be admitted with a diagnosis of STEMI (OR: 2.96, 95% CI: 1.85-4.72). Angiographic characteristics associated with no-reflow phenomenon were: stenosis >= 50% of the right coronary artery, presence of multivessel disease and pre-procedural TIMI blood flow grades 0-1. No-reflow was highly predictive of in-hospital mortality (17.2% vs. 4.2%; adjusted OR: 4.60, 95% CI: 2.61-8.09). Administration of pre-procedural unfractioned heparin or 600 mg clopidogrel loading dose was associated with less incidence of no-reflow (OR: 0.65, 95% CI: 0.43-0.99 and 0.61, 95% CI: 0.37-1.00, respectively). Aspirin, enoxaparin, and 300 mg clopidogrel loading dose, did not significantly impact the occurrence of the no-reflow. Conclusions: We found that pre-procedural administration of 600 mg loading dose of clopidogrel and/or unfractioned heparin is associated with reduced incidence of no-reflow. (C) 2016 Elsevier Ireland Ltd. All rights reserved.

Filiaciones:
Cenko, E:
 Univ Bologna, Dept Expt Diagnost & Specialty Med, I-40126 Bologna, Italy

Ricci, B:
 Univ Bologna, Dept Expt Diagnost & Specialty Med, I-40126 Bologna, Italy

Kedev, S:
 Univ St Cyril & Methodius, Univ Clin Cardiol, Fac Med, Skopje, North Macedonia

Kalpak, O:
 Univ St Cyril & Methodius, Univ Clin Cardiol, Fac Med, Skopje, North Macedonia

Calmac, L:
 Clin Emergency Hosp Bucharest, Dept Cardiol, Bucharest, Romania

Vasiljevic, Z:
 Univ Belgrade, Clin Ctr Serbia, Belgrade, Serbia

Knezevic, B:
 Clin Ctr Montenegro, Ctr Cardiol, Podgorica, Montenegro

Dilic, M:
 Univ Sarajevo, Ctr Clin, Sarajevo, Bosnia & Herceg

Milicic, D:
 Univ Zagreb, Univ Hosp Ctr Zagreb, Dept Cardiovasc Dis, Zagreb, Croatia

Manfrini, O:
 Univ Bologna, Dept Expt Diagnost & Specialty Med, I-40126 Bologna, Italy

Koller, A:
 Univ Phys Educ, Inst Nat Sci, H-1123 Budapest, Hungary

Dorobantu, M:
 Clin Emergency Hosp Bucharest, Dept Cardiol, Bucharest, Romania

 Bucharest Univ Med & Pharm Carol Davila, Bucharest, Romania

Badimon, L:
 Autonomous Univ Barcelona, Inst Carlos III, Cardiovasc Res Ctr, CSIC,ICCC,Hosp Santa Creu & St Pau, Barcelona, Spain

Bugiardini, R:
 Univ Bologna, Dept Expt Diagnost & Specialty Med, I-40126 Bologna, Italy
ISSN: 01675273





INTERNATIONAL JOURNAL OF CARDIOLOGY
Editorial
ELSEVIER IRELAND LTD, ELSEVIER HOUSE, BROOKVALE PLAZA, EAST PARK SHANNON, CO, CLARE, 00000, IRELAND, Irlanda
Tipo de documento: Article
Volumen: 222 Número:
Páginas: 1122-1128
WOS Id: 000384698300243
ID de PubMed: 27499222

MÉTRICAS