Neuronal exosomes reveal Alzheimer's disease biomarkers in Down syndrome


Por: Hamlett, ED, Goetzl, EJ, Ledreux, A, Vasilevko, V, Boger, HA, LaRosa, A, Clark, D, Carroll, SL, Carmona-Iragui, M, Fortea, J, Mufson, EJ, Sabbagh, M, Mohammed, AH, Hartley, D, Doran, E, Lott, IT, Granholm, AC

Publicada: 1 may 2017
Resumen:
Introduction: Individuals with Down syndrome (DS) exhibit Alzheimer's disease (AD) neuropathology and dementia early in life. Blood biomarkers of AD neuropathology would be valuable, as non-AD intellectual disabilities of DS and AD dementia overlap clinically. We hypothesized that elevations of amyloid beta (A beta) peptides and phosphorylated-tau in neuronal exosomes may document preclinical AD. Methods: AD neuropathogenic proteins A beta(1-42), P-T181-tau, and P-S396-tau were quantified by enzyme-linked immunosorbent assays in extracts of neuronal exosomes purified from blood of individuals with DS and age-matched controls. Results: Neuronal exosome levels of A beta(1-42), P-T181-tau, and P-S396-tau were significantly elevated in individuals with DS compared with age-matched controls at all ages beginning in childhood. No significant gender differences were observed. Discussion: These early increases in A beta(1-42), P-T181-tau, and P-S396-tau in individuals with DS may provide a basis for early intervention as targeted treatments become available. (C) 2016 Published by Elsevier Inc. on behalf of the Alzheimer's Association.

Filiaciones:
Hamlett, ED:
 Med Univ South Carolina, Dept Neurosci, Charleston, SC 29425 USA

 Univ Denver, Knoebel Inst Hlth Aging, Denver, CO 80208 USA

Goetzl, EJ:
 Jewish Home San Francisco, Geriatr Res Ctr, San Francisco, CA USA

 Univ Calif San Francisco, Dept Med, San Francisco, CA USA

Ledreux, A:
 Med Univ South Carolina, Dept Neurosci, Charleston, SC 29425 USA

 Univ Denver, Knoebel Inst Hlth Aging, Denver, CO 80208 USA

Vasilevko, V:
 Univ Calif Irvine, Irvine Inst Memory Impairment & Neurol Disorders, Irvine, CA USA

Boger, HA:
 Med Univ South Carolina, Dept Neurosci, Charleston, SC 29425 USA

 Med Univ South Carolina, Dept Neurosci, Ctr Aging, Charleston, SC 29425 USA

LaRosa, A:
 Med Univ South Carolina, Dept Pediat, Charleston, SC USA

Clark, D:
 Med Univ South Carolina, Dept Neurol, Charleston, SC USA

Carroll, SL:
 Med Univ South Carolina, Dept Pathol & Lab Med, Charleston, SC USA

Carmona-Iragui, M:
 Hosp Santa Creu & Sant Pau, Biomed Res Inst St Pau, Memory Unit, Neurol Dept, Barcelona, Spain

 Fundacio Catalana Sindrome Down, Down Med Ctr, Barcelona, Spain

Fortea, J:
 Hosp Santa Creu & Sant Pau, Biomed Res Inst St Pau, Memory Unit, Neurol Dept, Barcelona, Spain

 Fundacio Catalana Sindrome Down, Down Med Ctr, Barcelona, Spain

Mufson, EJ:
 Barrow Neurol Inst, Dept Neurobiol, Phoenix, AZ 85013 USA

Sabbagh, M:
 Barrow Neurol Inst, Dept Neurobiol, Phoenix, AZ 85013 USA

Mohammed, AH:
 Linnaeus Univ, Dept Psychol, Vaxjo, Sweden

 Karolinska Inst, Ctr Alzheimer Res, Huddinge, Sweden

Hartley, D:
 Alzheimers Assoc, Chicago, IL USA

Doran, E:
 Univ Calif Irvine, Dept Pediat, Sch Med, Orange, CA 92717 USA

Lott, IT:
 Barrow Neurol Inst, Dept Neurobiol, Phoenix, AZ 85013 USA

 Univ Calif Irvine, Dept Pediat, Sch Med, Orange, CA 92717 USA

Granholm, AC:
 Med Univ South Carolina, Dept Neurosci, Charleston, SC 29425 USA

 Univ Denver, Knoebel Inst Hlth Aging, Denver, CO 80208 USA

 Med Univ South Carolina, Dept Neurosci, Ctr Aging, Charleston, SC 29425 USA

 Barrow Neurol Inst, Dept Neurobiol, Phoenix, AZ 85013 USA

 Karolinska Inst, Ctr Alzheimer Res, Huddinge, Sweden
ISSN: 15525260
Editorial
WILEY, 111 RIVER ST, HOBOKEN 07030-5774, NJ USA, Estados Unidos America
Tipo de documento: Article
Volumen: 13 Número: 5
Páginas: 541-549
WOS Id: 000400554400005
ID de PubMed: 27755974
imagen Green Accepted

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