Phase II trial of dacomitinib, a pan-human EGFR tyrosine kinase inhibitor, in recurrent glioblastoma patients with EGFR amplification


Por: Sepulveda-Sanchez, JM, Vaz, MA, Balana, C, Gil-Gil, M, Reynes, G, Gallego, O, Martinez-Garcia, M, Vicente, E, Quindos, M, Luque, R, Ramos, A, Ruano, Y, Perez-Segura, P, Benavides, M, Sanchez-Gomez, P, Hernandez-Lain, A

Publicada: 1 nov 2017
Resumen:
Background. We conducted a multicenter, 2-stage, open-label, phase II trial to assess the efficacy and safety of dacomitinib in adult patients with recurrent glioblastoma (GB) and epidermal growth factor receptor gene (EGFR) amplification with or without variant III (EGFRvIII) deletion. Methods. Patients with first recurrence were enrolled in 2 cohorts. Cohort A included patients with EGFR gene amplification without EGFRvIII mutation. Cohort B included patients with EGFR gene amplification and EGFRvIII mutation. Dacomitinib was administered (45 mg/day) until disease progression/unacceptable adverse events (AEs). Primary endpoint was progression-free survival (PFS; RANO criteria) at 6 months (PFS6). Results. Thirty patients in Cohort A and 19 in Cohort B were enrolled. Median age was 59 years (range 39-81), 65.3% were male, and Eastern Cooperative Oncology Group Performance Status 0/1/2 were 10.2% /65.3% /24.5%, respectively. PFS6 was 10.6% (Cohort A: 13.3%; Cohort B: 5.9%) with a median PFS of 2.7 months (Cohort A: 2.7 mo; Cohort B: 2.6 mo). Four patients were progression free at 6 months and 3 patients were so at 12 months. Median overall survival was 7.4 months (Cohort A: 7.8 mo; Cohort B: 6.7 mo). The best overall response included 1 complete response and 2 partial responses (4.1%). Stable disease was observed in 12 patients (24.5%: eight in Cohort A and four in Cohort B). Diarrhea and rash were the most common AEs; 20 (40.8%) patients experienced grade 3-4 drug-related AEs. Conclusions. Dacomitinib has a limited single-agent activity in recurrent GB with EGFR amplification. The detailed molecular characterization of the 4 patients with response in this trial can be useful to select patients who could benefit from dacomitinib.

Filiaciones:
Sepulveda-Sanchez, JM:
 Hosp Univ 12 Octubre, Neurooncol Unit, Madrid, Spain

Vaz, MA:
 Hosp Univ Ramon & Cajal, Med Oncol, Madrid, Spain

Balana, C:
 Inst Catala Oncol, Med Oncol, Badalona, Spain

Gil-Gil, M:
 IDIBELL, Inst Catala Oncol, Med Oncol, Barcelona, Spain

Reynes, G:
 Hosp Univ & Politecn La Fe, Med Oncol, Valencia, Spain

Gallego, O:
 Univ Autonoma Barcelona, Hosp Santa Creu & St Pau, Med Oncol, Barcelona, Spain

Martinez-Garcia, M:
 Hosp del Mar, Med Oncol, Barcelona, Spain

Vicente, E:
 Hosp Univ Insular Gran Canaria, Med Oncol, Las Palmas Gran Canaria, Spain

Quindos, M:
 Hosp A Coruna, Med Oncol, La Coruna, Spain

Luque, R:
 Complejo Hosp Granada, Med Oncol, Granada, Spain

Ramos, A:
 Hosp Univ 12 Octubre, Neuroradiol, Madrid, Spain

Ruano, Y:
 Hosp Univ 12 Octubre, Mol Pathol Unit, Madrid, Spain

Perez-Segura, P:
 Hosp Clin San Carlos, Med Oncol, Madrid, Spain

Benavides, M:
 Hosp Univ Reg & Virgen de la Victoria, Med Oncol, Malaga, Spain

Sanchez-Gomez, P:
 Inst Salud Carlos III, Neurooncol Unit, Madrid, Spain

Hernandez-Lain, A:
 Hosp Univ 12 Octubre, Neuropathol, Madrid, Spain
ISSN: 15228517





NEURO-ONCOLOGY
Editorial
OXFORD UNIV PRESS INC, JOURNALS DEPT, 2001 EVANS RD, CARY, NC 27513 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 19 Número: 11
Páginas: 1522-1531
WOS Id: 000413219400013
ID de PubMed: 28575464
imagen Green Published, Bronze

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