Distal hereditary motor neuropathies: Mutation spectrum and genotype-phenotype correlation


Por: Frasquet, M, Rojas-Garcia, R, Argente-Escrig, H, Vazquez-Costa, JF, Muelas, N, Vilchez, JJ, Sivera, R, Millet, E, Barreiro, M, Diaz-Manera, J, Turon-Sans, J, Cortes-Vicente, E, Querol, L, Ramirez-Jimenez, L, Martinez-Rubio, D, Sanchez-Monteagudo, A, Espinos, C, Sevilla, T, Lupo, V

Publicada: 1 abr 2021 Ahead of Print: 1 ene 2021
Resumen:
Background and purpose Distal hereditary motor neuropathies (dHMNs) are a heterogeneous group of disorders characterized by degeneration of the motor component of peripheral nerves. Currently, only 15% to 32.5% of patients with dHMN are characterized genetically. Additionally, the prevalence of these genetic disorders is not well known. Recently, biallelic mutations in the sorbitol dehydrogenase gene (SORD) have been identified as a cause of dHMN, with an estimated frequency in undiagnosed cases of up to 10%. Methods In the present study, we included 163 patients belonging to 108 different families who were diagnosed with a dHMN and who underwent a thorough genetic screening that included next-generation sequencing and subsequent Sanger sequencing of SORD. Results Most probands were sporadic cases (62.3%), and the most frequent age of onset of symptoms was 2 to 10 years (28.8%). A genetic diagnosis was achieved in 37/108 (34.2%) families and 78/163 (47.8%) of all patients. The most frequent cause of distal hereditary motor neuropathies were mutations in HSPB1 (10.4%), GARS1 (9.8%), BICD2 (8.0%), and DNAJB2 (6.7%) genes. In addition, 3.1% of patients were found to be carriers of biallelic mutations in SORD. Mutations in another seven genes were also identified, although they were much less frequent. Eight new pathogenic mutations were detected, and 17 patients without a definite genetic diagnosis carried variants of uncertain significance. The calculated minimum prevalence of dHMN was 2.3 per 100,000 individuals. Conclusions This study confirms the genetic heterogeneity of dHMN and that biallelic SORD mutations are a cause of dHMN in different populations.

Filiaciones:
Frasquet, M:
 Hosp Univ & Politecn La Fe, Dept Neurol, Neuromuscular Dis Unit, Valencia, Spain

 Inst Invest Sanitaria La Fe, Neuromuscular & Ataxias Res Grp, Valencia, Spain

 Ctr Invest Biomed Red Enfermedades Raras CIBERER, Valencia, Spain

Rojas-Garcia, R:
 Ctr Invest Biomed Red Enfermedades Raras CIBERER, Valencia, Spain

 Hosp Santa Creu & Sant Pau, Dept Neurol, Neuromuscular Dis Unit, Barcelona, Spain

 Univ Autonoma Barcelona, Barcelona, Spain

Argente-Escrig, H:
 Inst Invest Sanitaria La Fe, Neuromuscular & Ataxias Res Grp, Valencia, Spain

 Ctr Invest Biomed Red Enfermedades Raras CIBERER, Valencia, Spain

Vazquez-Costa, JF:
 Hosp Univ & Politecn La Fe, Dept Neurol, Neuromuscular Dis Unit, Valencia, Spain

 Inst Invest Sanitaria La Fe, Neuromuscular & Ataxias Res Grp, Valencia, Spain

 Ctr Invest Biomed Red Enfermedades Raras CIBERER, Valencia, Spain

 Univ Valencia, Dept Med, Valencia, Spain

Muelas, N:
 Hosp Univ & Politecn La Fe, Dept Neurol, Neuromuscular Dis Unit, Valencia, Spain

 Inst Invest Sanitaria La Fe, Neuromuscular & Ataxias Res Grp, Valencia, Spain

 Ctr Invest Biomed Red Enfermedades Raras CIBERER, Valencia, Spain

Vilchez, JJ:
 Inst Invest Sanitaria La Fe, Neuromuscular & Ataxias Res Grp, Valencia, Spain

 Ctr Invest Biomed Red Enfermedades Raras CIBERER, Valencia, Spain

Sivera, R:
 Hosp Francesc Borja, Dept Neurol, Gandia, Spain

Millet, E:
 Hosp Univ & Politecn La Fe, Dept Clin Neurophysiol, Valencia, Spain

Barreiro, M:
 Inst Invest Sanitaria La Fe, Neuromuscular & Ataxias Res Grp, Valencia, Spain

Diaz-Manera, J:
 Ctr Invest Biomed Red Enfermedades Raras CIBERER, Valencia, Spain

 Hosp Santa Creu & Sant Pau, Dept Neurol, Neuromuscular Dis Unit, Barcelona, Spain

 Univ Autonoma Barcelona, Barcelona, Spain

Turon-Sans, J:
 Ctr Invest Biomed Red Enfermedades Raras CIBERER, Valencia, Spain

 Hosp Santa Creu & Sant Pau, Dept Neurol, Neuromuscular Dis Unit, Barcelona, Spain

 Univ Autonoma Barcelona, Barcelona, Spain

Cortes-Vicente, E:
 Ctr Invest Biomed Red Enfermedades Raras CIBERER, Valencia, Spain

 Hosp Santa Creu & Sant Pau, Dept Neurol, Neuromuscular Dis Unit, Barcelona, Spain

 Univ Autonoma Barcelona, Barcelona, Spain

Querol, L:
 Ctr Invest Biomed Red Enfermedades Raras CIBERER, Valencia, Spain

 Hosp Santa Creu & Sant Pau, Dept Neurol, Neuromuscular Dis Unit, Barcelona, Spain

 Univ Autonoma Barcelona, Barcelona, Spain

Ramirez-Jimenez, L:
 Ctr Invest Principe Felipe CIPF, Dept Genom & Translat Genet, Valencia, Spain

Martinez-Rubio, D:
 Ctr Invest Principe Felipe CIPF, Unit Genet & Genom Neuromuscular & Neurodegenerat, Valencia, Spain

 INCLIVA & IIS La Fe CIPF, Rare Dis Joint Units, Valencia, Spain

Sanchez-Monteagudo, A:
 Ctr Invest Principe Felipe CIPF, Unit Genet & Genom Neuromuscular & Neurodegenerat, Valencia, Spain

 INCLIVA & IIS La Fe CIPF, Rare Dis Joint Units, Valencia, Spain

Espinos, C:
 Ctr Invest Principe Felipe CIPF, Unit Genet & Genom Neuromuscular & Neurodegenerat, Valencia, Spain

 INCLIVA & IIS La Fe CIPF, Rare Dis Joint Units, Valencia, Spain

Sevilla, T:
 Hosp Univ & Politecn La Fe, Dept Neurol, Neuromuscular Dis Unit, Valencia, Spain

 Inst Invest Sanitaria La Fe, Neuromuscular & Ataxias Res Grp, Valencia, Spain

 Ctr Invest Biomed Red Enfermedades Raras CIBERER, Valencia, Spain

 Univ Valencia, Dept Med, Valencia, Spain

Lupo, V:
 Ctr Invest Principe Felipe CIPF, Unit Genet & Genom Neuromuscular & Neurodegenerat, Valencia, Spain

 INCLIVA & IIS La Fe CIPF, Rare Dis Joint Units, Valencia, Spain
ISSN: 13515101





EUROPEAN JOURNAL OF NEUROLOGY
Editorial
WILEY, 111 RIVER ST, HOBOKEN 07030-5774, NJ USA, Reino Unido
Tipo de documento: Article
Volumen: 28 Número: 4
Páginas: 1334-1343
WOS Id: 000606334600001
ID de PubMed: 33369814

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