Integrated Multiomics Reveals Glucose Use Reprogramming and Identifies a Novel Hexokinase in Alcoholic Hepatitis


Por: Massey, V, Parrish, A, Argemi, J, Moreno, M, Mello, A, Garcia-Rocha, M, Altamirano, J, Odena, G, Dubuquoy, L, Louvet, A, Martinez, C, Adrover, A, Affo, S, Morales-Ibanez, O, Sancho-Bru, P, Millan, C, Alvarado-Tapias, E, Morales-Arraez, D, Caballeria, J, Mann, J, Cao, S, Sun, ZL, Shah, V, Cameron, A, Mathurin, P, Snider, N, Villanueva, C, Morgan, TR, Guinovart, J, Vadigepalli, R, Bataller, R

Publicada: 1 mar 2021 Ahead of Print: 1 abr 2021
Resumen:
BACKGROUND & AIMS: We recently showed that alcoholic hepatitis (AH) is characterized by dedifferentiation of hepatocytes and loss of mature functions. Glucose metabolism is tightly regulated in healthy hepatocytes. We hypothesize that AH may lead to metabolic reprogramming of the liver, including dysregulation of glucose metabolism. METHODS: We performed integrated metabolomic and transcriptomic analyses of liver tissue from patients with AH or alcoholic cirrhosis or normal liver tissue from hepatic resection. Focused analyses of chromatin immunoprecipitation coupled to DNA sequencing was performed. Functional in vitro studies were performed in primary rat and human hepatocytes and HepG2 cells. RESULTS: Patients with AH exhibited specific changes in the levels of intermediates of glycolysis/gluconeogenesis, the tricarboxylic acid cycle, and monosaccharide and disaccharide metabolism. Integrated analysis of the transcriptome and metabolome showed the used of alternate energetic pathways, metabolite sinks and bottlenecks, and dysregulated glucose storage in patients with AH. Among genes involved in glucose metabolism, hexokinase domain containing 1 (HKDC1) was identified as the most up-regulated kinase in patients with AH. Histone active promoter and enhancer markers were increased in the HKDC1 genomic region. High HKDC1 levels were associated with the development of acute kidney injury and decreased survival. Increased HKDC1 activity contributed to the accumulation of glucose-6-P and glycogen in primary rat hepatocytes. CONCLUSIONS: Altered metabolite levels and messenger RNA expression of metabolic enzymes suggest the existence of extensive reprogramming of glucose metabolism in AH. Increased HKDC1 expression may contribute to dysregulated glucose metabolism and represents a novel biomarker and therapeutic target for AH.

Filiaciones:
Massey, V:
 Univ N Carolina, Dept Med, Div Gastroenterol & Hepatol, Chapel Hill, NC 27515 USA

 Univ N Carolina, Dept Nutr, Div Gastroenterol & Hepatol, Chapel Hill, NC 27515 USA

 Univ N Carolina, Bowles Ctr Alcohol Studies, Chapel Hill, NC 27515 USA

Parrish, A:
 Thomas Jefferson Univ, Dept Pathol Anat & Cell Biol, Daniel Baugh Inst, Philadelphia, PA 19107 USA

Argemi, J:
 Univ Pittsburgh, Med Ctr, Div Med, Dept Gastroenterol & Hepatol, Pittsburgh, PA USA

 Clin Univ Navarra, Liver Unit, Pamplona, Spain

 IdisNA, Ctr Appl Med Res, Hepatol Program, Pamplona, Spain

Moreno, M:
 Inst Invest Biomed August Pi i Sunyer, Barcelona, Spain

Mello, A:
 Univ Pittsburgh, Med Ctr, Div Med, Dept Gastroenterol & Hepatol, Pittsburgh, PA USA

Garcia-Rocha, M:
 Barcelona Inst Sci & Technol, Inst Res Biomed, Barcelona, Spain

Altamirano, J:
 Inst Invest Biomed August Pi i Sunyer, Barcelona, Spain

 Hosp Univ Vall dHebron, Vall dHebron Inst Recerca, Internal Med Dept, Liver Unit, Barcelona, Spain

Odena, G:
 Univ N Carolina, Dept Med, Div Gastroenterol & Hepatol, Chapel Hill, NC 27515 USA

 Univ N Carolina, Dept Nutr, Div Gastroenterol & Hepatol, Chapel Hill, NC 27515 USA

 Univ N Carolina, Bowles Ctr Alcohol Studies, Chapel Hill, NC 27515 USA

Dubuquoy, L:
 Univ Lille, Inserm LIRIC UMR995, CHU Lille, Serv Malad Appareil Digestif, Lille, France

Louvet, A:
 Univ Lille, Inserm LIRIC UMR995, CHU Lille, Serv Malad Appareil Digestif, Lille, France

Martinez, C:
 Barcelona Inst Sci & Technol, Inst Res Biomed, Barcelona, Spain

Adrover, A:
 Barcelona Inst Sci & Technol, Inst Res Biomed, Barcelona, Spain

Affo, S:
 Inst Invest Biomed August Pi i Sunyer, Barcelona, Spain

Morales-Ibanez, O:
 Inst Invest Biomed August Pi i Sunyer, Barcelona, Spain

Sancho-Bru, P:
 Inst Invest Biomed August Pi i Sunyer, Barcelona, Spain

Millan, C:
 Inst Invest Biomed August Pi i Sunyer, Barcelona, Spain

Alvarado-Tapias, E:
 Hosp Santa Creu & Sant Pau, Dept Gastroenterol, Barcelona, Spain

 Inst Salud Carlos III, Ctr Invest Biomed Red Enfermedades Hepat & Digest, Madrid, Spain

Morales-Arraez, D:
 Univ Pittsburgh, Med Ctr, Div Med, Dept Gastroenterol & Hepatol, Pittsburgh, PA USA

Caballeria, J:
 Inst Invest Biomed August Pi i Sunyer, Barcelona, Spain

 Univ Barcelona, Fac Med, CIBER Enfermedades Hepat & Digest, Hosp Clin,Liver Unit, Barcelona, Spain

Mann, J:
 Newcastle Univ, Fac Med Sci, Inst Cellular Med, Newcastle Fibrosis Res Grp, Newcastle Upon Tyne, Tyne & Wear, England

Cao, S:
 Mayo Clin, Div Gastroenterol & Hepatol, Rochester, MN USA

Sun, ZL:
 Johns Hopkins Sch Med, Dept Surg, Baltimore, MD USA

 Johns Hopkins Sch Med, Transplant Biol Res Ctr, Baltimore, MD USA

Shah, V:
 Mayo Clin, Div Gastroenterol & Hepatol, Rochester, MN USA

Cameron, A:
 Johns Hopkins Sch Med, Dept Surg, Baltimore, MD USA

 Johns Hopkins Sch Med, Transplant Biol Res Ctr, Baltimore, MD USA

Mathurin, P:
 Univ Lille, Inserm LIRIC UMR995, CHU Lille, Serv Malad Appareil Digestif, Lille, France

Snider, N:
 Univ N Carolina, Dept Cell Biol & Physiol, Chapel Hill, NC 27515 USA

Villanueva, C:
 Hosp Santa Creu & Sant Pau, Dept Gastroenterol, Barcelona, Spain

 Inst Salud Carlos III, Ctr Invest Biomed Red Enfermedades Hepat & Digest, Madrid, Spain

 Hosp Santa Creu & Sant Pau, Inst Recerca, Barcelona, Spain

 Univ Autonoma Barcelona, Barcelona, Cerdanyola Del, Spain

Morgan, TR:
 VA Long Beach Healthcare Syst, VA Long Beach Healthcare, Gastroenterol Serv, Long Beach, CA USA

Guinovart, J:
 Barcelona Inst Sci & Technol, Inst Res Biomed, Barcelona, Spain

Vadigepalli, R:
 Thomas Jefferson Univ, Dept Pathol Anat & Cell Biol, Daniel Baugh Inst, Philadelphia, PA 19107 USA

Bataller, R:
 Univ N Carolina, Dept Med, Div Gastroenterol & Hepatol, Chapel Hill, NC 27515 USA

 Univ N Carolina, Dept Nutr, Div Gastroenterol & Hepatol, Chapel Hill, NC 27515 USA

 Univ N Carolina, Bowles Ctr Alcohol Studies, Chapel Hill, NC 27515 USA

 Univ Pittsburgh, Med Ctr, Div Med, Dept Gastroenterol & Hepatol, Pittsburgh, PA USA
ISSN: 00165085





GASTROENTEROLOGY
Editorial
W B SAUNDERS CO-ELSEVIER INC, 1600 JOHN F KENNEDY BOULEVARD, STE 1800, PHILADELPHIA, PA 19103-2899 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 160 Número: 5
Páginas: 1725
WOS Id: 000637453900037
ID de PubMed: 33309778
imagen Green Accepted

MÉTRICAS