Structure and Dynamics of Large-Scale Cognitive Networks in Huntington's Disease


Por: Aracil-Bolanos, I, Martinez-Horta, S, Gonzalez-de-Echavarri, JM, Sampedro, F, Perez-Perez, J, Horta, A, Campolongo, A, Izquierdo, C, Gomez-Anson, B, Pagonabarraga, J, Kulisevsky, J

Publicada: 1 feb 2022 Ahead of Print: 1 nov 2021
Resumen:
Background Huntington's disease is a neurodegenerative disorder characterized by clinical alterations in the motor, behavioral, and cognitive domains. However, the structure and disruptions to large-scale brain cognitive networks have not yet been established. Objective We aimed to profile changes in large-scale cognitive networks in premanifest and symptomatic patients with Huntington's disease. Methods We prospectively recruited premanifest and symptomatic Huntington's disease mutation carriers as well as healthy controls. Clinical and sociodemographic data were obtained from all participants, and resting-state functional connectivity data, using both time-averaged and dynamic functional connectivity, was acquired from whole-brain and cognitively oriented brain parcellations. Results A total of 64 gene mutation carriers and 23 healthy controls were included; 21 patients with Huntington's disease were classified as premanifest and 43 as symptomatic Huntington's disease. Compared with healthy controls, patients with Huntington's disease showed decreased network connectivity within the posterior hubs of the default-mode network and the medial prefrontal cortex, changes that correlated with cognitive (t = 2.25, P = 0.01) and disease burden scores (t = -2.42, P = 0.009). The salience network showed decreased functional connectivity between insular and supramarginal cortices and also correlated with cognitive (t = 2.11, P = 0.02) and disease burden scores (t = -2.35, P = 0.01). Dynamic analyses showed that network variability was decreased for default-central executive networks, a feature already present in premanifest mutation carriers (dynamic factor 8, P = 0.02). Conclusions Huntington's disease shows an early and widespread disruption of large-scale cognitive networks. Importantly, these changes are related to cognitive and disease burden scores, and novel dynamic functional analyses uncovered subtler network changes even in the premanifest stages.

Filiaciones:
Aracil-Bolanos, I:
 St Pau Hosp, Movement Disorders Unit, Neurol Dept, Mas Casanovas 90, Barcelona 08041, Spain

 Univ Autonoma Barcelona, Dept Med, Barcelona, Spain

 Inst Invest Biomed St Pau, Barcelona, Spain

 Ctr Invest Red Enfermedades Neurodegenerat, Madrid, Spain

Martinez-Horta, S:
 St Pau Hosp, Movement Disorders Unit, Neurol Dept, Mas Casanovas 90, Barcelona 08041, Spain

 Univ Autonoma Barcelona, Dept Med, Barcelona, Spain

 Inst Invest Biomed St Pau, Barcelona, Spain

 Ctr Invest Red Enfermedades Neurodegenerat, Madrid, Spain

Gonzalez-de-Echavarri, JM:
 Pasqual Maragall Fdn, Barcelonasseta Brain Res Ctr, Barcelona, Spain

 Hosp Mar Med Res Inst, Barcelona, Spain

Sampedro, F:
 St Pau Hosp, Movement Disorders Unit, Neurol Dept, Mas Casanovas 90, Barcelona 08041, Spain

 Univ Autonoma Barcelona, Dept Med, Barcelona, Spain

 Inst Invest Biomed St Pau, Barcelona, Spain

 Ctr Invest Red Enfermedades Neurodegenerat, Madrid, Spain

Perez-Perez, J:
 St Pau Hosp, Movement Disorders Unit, Neurol Dept, Mas Casanovas 90, Barcelona 08041, Spain

 Univ Autonoma Barcelona, Dept Med, Barcelona, Spain

 Inst Invest Biomed St Pau, Barcelona, Spain

 Ctr Invest Red Enfermedades Neurodegenerat, Madrid, Spain

Horta, A:
 St Pau Hosp, Movement Disorders Unit, Neurol Dept, Mas Casanovas 90, Barcelona 08041, Spain

 Univ Autonoma Barcelona, Dept Med, Barcelona, Spain

 Inst Invest Biomed St Pau, Barcelona, Spain

 Ctr Invest Red Enfermedades Neurodegenerat, Madrid, Spain

Campolongo, A:
 St Pau Hosp, Movement Disorders Unit, Neurol Dept, Mas Casanovas 90, Barcelona 08041, Spain

 Univ Autonoma Barcelona, Dept Med, Barcelona, Spain

 Inst Invest Biomed St Pau, Barcelona, Spain

 Ctr Invest Red Enfermedades Neurodegenerat, Madrid, Spain

Izquierdo, C:
 St Pau Hosp, Movement Disorders Unit, Neurol Dept, Mas Casanovas 90, Barcelona 08041, Spain

 Univ Autonoma Barcelona, Dept Med, Barcelona, Spain

 Inst Invest Biomed St Pau, Barcelona, Spain

 Ctr Invest Red Enfermedades Neurodegenerat, Madrid, Spain

Gomez-Anson, B:
 Univ Autonoma Barcelona, Dept Med, Barcelona, Spain

 Inst Invest Biomed St Pau, Barcelona, Spain

 Ctr Invest Red Enfermedades Neurodegenerat, Madrid, Spain

 St Pau Hosp, Neuroradiol Unit, Barcelona, Spain

Pagonabarraga, J:
 St Pau Hosp, Movement Disorders Unit, Neurol Dept, Mas Casanovas 90, Barcelona 08041, Spain

 Univ Autonoma Barcelona, Dept Med, Barcelona, Spain

 Inst Invest Biomed St Pau, Barcelona, Spain

 Ctr Invest Red Enfermedades Neurodegenerat, Madrid, Spain

Kulisevsky, J:
 St Pau Hosp, Movement Disorders Unit, Neurol Dept, Mas Casanovas 90, Barcelona 08041, Spain

 Univ Autonoma Barcelona, Dept Med, Barcelona, Spain

 Inst Invest Biomed St Pau, Barcelona, Spain

 Ctr Invest Red Enfermedades Neurodegenerat, Madrid, Spain
ISSN: 08853185
Editorial
WILEY, 111 RIVER ST, HOBOKEN 07030-5774, NJ USA, USA
Tipo de documento: Article
Volumen: 37 Número: 2
Páginas: 343-353
WOS Id: 000715906800001
ID de PubMed: 34752656
imagen hybrid, All Open Access, Hybrid Gold

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