Mutations in TOP3A Cause a Bloom Syndrome-like Disorder


Por: Martin, CA, Sarlos, K, Logan, CV, Thakur, RS, Parry, DA, Bizard, AH, Leitch, A, Cleal, L, Ali, NS, Al-Owain, MA, Allen, W, Altmueller, J, Aza-Carmona, M, Barakat, BAY, Barraza-Garcia, J, Begtrup, A, Bogliolo, M, Cho, MT, Cruz-Rojo, J, Dhahrabi, HAM, Elcioglu, NH, Gosgene, Gorman, GS, Jobling, R, Kesterton, I, Kishita, Y, Kohda, M, Stabej, PLQ, Malallah, AJ, Nuernberg, P, Ohtake, A, Okazaki, Y, Pujol, R, Ramirez, MJ, Revah-Politi, A, Shimura, M, Stevens, P, Taylor, RW, Turner, L, Williams, H, Wilson, C, Yigit, G, Zahavich, L, Alkuraya, FS, Surralles, J, Iglesais, A, Murayama, K, Wollnik, B, Dattani, M, Heath, KE, Hickson, ID, Jackson, AP

Publicada: 2 ago 2018
Resumen:
Bloom syndrome, caused by biallelic mutations in BLM, is characterized by prenatal-onset growth deficiency, short stature, an erythematous photosensitive malar rash, and increased cancer predisposition. Diagnostically, a hallmark feature is the presence of increased sister chromatid exchanges (SCEs) on cytogenetic testing. Here, we describe biallelic mutations in TOP3A in ten individuals with prenatal-onset growth restriction and microcephaly. TOP3A encodes topoisomerase III alpha (TopIII alpha), which binds to BLM as part of the BTRR complex, and promotes dissolution of double Holliday junctions arising during homologous recombination. We also identify a homozygous truncating variant in RMI1, which encodes another component of the BTRR complex, in two individuals with microcephalic dwarfism. The TOP3A mutations substantially reduce cellular levels of TopIII alpha, and consequently subjects' cells demonstrate elevated rates of SCE. Unresolved DNA recombination and/or replication intermediates persist into mitosis, leading to chromosome segregation defects and genome instability that most likely explain the growth restriction seen in these subjects and in Bloom syndrome. Clinical features of mitochondrial dysfunction are evident in several individuals with biallelic TOP3A mutations, consistent with the recently reported additional function of TopIII alpha in mitochondrial DNA decatenation. In summary, our findings establish TOP3A mutations as an additional cause of prenatal-onset short stature with increased cytogenetic SCEs and implicate the decatenation activity of the BTRR complex in their pathogenesis.

Filiaciones:
Martin, CA:
 Univ Edinburgh, MRC Inst Genet & Mol Med, MRC Human Genet Unit, Edinburgh EH4 2XU, Midlothian, Scotland

Sarlos, K:
 Univ Copenhagen, Dept Cellular & Mol Med, Ctr Chromosome Stabil & Ctr Hlth, Blegdamsvej 3B, DK-2200 Copenhagen N, Denmark

Logan, CV:
 Univ Edinburgh, MRC Inst Genet & Mol Med, MRC Human Genet Unit, Edinburgh EH4 2XU, Midlothian, Scotland

Thakur, RS:
 Univ Copenhagen, Dept Cellular & Mol Med, Ctr Chromosome Stabil & Ctr Hlth, Blegdamsvej 3B, DK-2200 Copenhagen N, Denmark

Parry, DA:
 Univ Edinburgh, MRC Inst Genet & Mol Med, MRC Human Genet Unit, Edinburgh EH4 2XU, Midlothian, Scotland

Bizard, AH:
 Univ Copenhagen, Dept Cellular & Mol Med, Ctr Chromosome Stabil & Ctr Hlth, Blegdamsvej 3B, DK-2200 Copenhagen N, Denmark

Leitch, A:
 Univ Edinburgh, MRC Inst Genet & Mol Med, MRC Human Genet Unit, Edinburgh EH4 2XU, Midlothian, Scotland

Cleal, L:
 Univ Edinburgh, MRC Inst Genet & Mol Med, MRC Human Genet Unit, Edinburgh EH4 2XU, Midlothian, Scotland

Ali, NS:
 Dubai Hosp, Khaleej St,Al Baraha,Box 7272, Dubai, U Arab Emirates

Al-Owain, MA:
 King Faisal Specialist Hosp & Res Ctr, Dept Med Genet, Riyadh 11211, Saudi Arabia

Allen, W:
 Fullerton Genet Ctr, Asheville, NC 28803 USA

Altmueller, J:
 Univ Cologne, Cologne Ctr Genom, D-50931 Cologne, Germany

Aza-Carmona, M:
 Univ Autonoma Madrid, Hosp Univ La Paz, Inst Med & Mol Genet, IdiPaz, Madrid 28046, Spain

 Univ Autonoma Madrid, Hosp Univ La Paz, Skeletal Dysplasia Multidisciplinary Unit, IdiPaz, Madrid 28046, Spain

 Ctr Invest Biomed Red Enfermedades Raras, Madrid 28029, Spain

Barakat, BAY:
 Dubai Hosp, Khaleej St,Al Baraha,Box 7272, Dubai, U Arab Emirates

Barraza-Garcia, J:
 Univ Autonoma Madrid, Hosp Univ La Paz, Inst Med & Mol Genet, IdiPaz, Madrid 28046, Spain

 Univ Autonoma Madrid, Hosp Univ La Paz, Skeletal Dysplasia Multidisciplinary Unit, IdiPaz, Madrid 28046, Spain

 Ctr Invest Biomed Red Enfermedades Raras, Madrid 28029, Spain

Begtrup, A:
 GeneDx, 207 Perry Pkwy, Gaithersburg, MD 20877 USA

Bogliolo, M:
 Ctr Invest Biomed Red Enfermedades Raras, Madrid 28029, Spain

 Univ Autonoma Barcelona, Dept Genet & Microbiol, Bellaterra 08193, Spain

Cho, MT:
 GeneDx, 207 Perry Pkwy, Gaithersburg, MD 20877 USA

Cruz-Rojo, J:
 Hosp 12 Octubre, Dept Pediat Endocrinol & Dysmorphol, E-28041 Madrid, Spain

Dhahrabi, HAM:
 Dubai Hosp, Khaleej St,Al Baraha,Box 7272, Dubai, U Arab Emirates

Elcioglu, NH:
 Marmara Univ, Sch Med, Dept Pediat Genet, TR-34722 Istanbul, Turkey

Gosgene:
 UCL, Great Ormond St Inst Child Hlth, 30 Guilford St, London WC1N 1EH, England

Gorman, GS:
 Newcastle Univ, Sch Med Educ, Inst Neurosci, Wellcome Ctr Mitochondrial Res, Newcastle Upon Tyne, Tyne & Wear, England

Jobling, R:
 Hosp Sick Children, Toronto, ON M5G 1X8, Canada

Kesterton, I:
 Guys Hosp, Viapath Analyt, Cytogenet Dept, London SE1 9RT, England

Kishita, Y:
 Juntendo Univ, Grad Sch Med, Intractable Dis Res Ctr, Bunkyo Ku, 2-1-1 Hongo, Tokyo 1138421, Japan

Kohda, M:
 Juntendo Univ, Grad Sch Med, Intractable Dis Res Ctr, Bunkyo Ku, 2-1-1 Hongo, Tokyo 1138421, Japan

Stabej, PLQ:
 UCL, Great Ormond St Inst Child Hlth, 30 Guilford St, London WC1N 1EH, England

Malallah, AJ:
 Dubai Hosp, Khaleej St,Al Baraha,Box 7272, Dubai, U Arab Emirates

Nuernberg, P:
 Univ Cologne, Cologne Ctr Genom, D-50931 Cologne, Germany

Ohtake, A:
 Saitama Med Univ, Fac Med, Dept Pediat, 38 Morohongo, Moroyama, Saitama 3500495, Japan

Okazaki, Y:
 Juntendo Univ, Grad Sch Med, Intractable Dis Res Ctr, Bunkyo Ku, 2-1-1 Hongo, Tokyo 1138421, Japan

Pujol, R:
 Ctr Invest Biomed Red Enfermedades Raras, Madrid 28029, Spain

 Univ Autonoma Barcelona, Dept Genet & Microbiol, Bellaterra 08193, Spain

Ramirez, MJ:
 Ctr Invest Biomed Red Enfermedades Raras, Madrid 28029, Spain

 Univ Autonoma Barcelona, Dept Genet & Microbiol, Bellaterra 08193, Spain

Revah-Politi, A:
 Columbia Univ, Med Ctr, Inst Genom Med, New York, NY 10032 USA

Shimura, M:
 Chiba Childrens Hosp, Dept Metab, Ctr Med Genet, Midori Ku, 579-1 Heta Cho, Chiba 2660007, Japan

Stevens, P:
 Guys Hosp, Viapath Analyt, Cytogenet Dept, London SE1 9RT, England

Taylor, RW:
 Newcastle Univ, Sch Med Educ, Inst Neurosci, Wellcome Ctr Mitochondrial Res, Newcastle Upon Tyne, Tyne & Wear, England

Turner, L:
 Mem Univ Newfoundland, St John, NF A1C 5S7, Canada

Williams, H:
 UCL, Great Ormond St Inst Child Hlth, 30 Guilford St, London WC1N 1EH, England

Wilson, C:
 Fullerton Genet Ctr, Asheville, NC 28803 USA

Yigit, G:
 Univ Med Ctr Gottingen, Inst Human Genet, D-37073 Gottingen, Germany

Zahavich, L:
 Hosp Sick Children, Toronto, ON M5G 1X8, Canada

Alkuraya, FS:
 King Faisal Specialist Hosp & Res Ctr, Dept Genet, Riyadh 11211, Saudi Arabia

Surralles, J:
 Ctr Invest Biomed Red Enfermedades Raras, Madrid 28029, Spain

 Univ Autonoma Barcelona, Dept Genet & Microbiol, Bellaterra 08193, Spain

 Hosp Santa Creu & Sant Pau, Dept Genet, Barcelona 08041, Spain

 Hosp Santa Creu & Sant Pau, Biomed Res Inst Sant Pau, Barcelona 08041, Spain

Iglesais, A:
 Columbia Univ, Med Ctr, Div Clin Genet, Dept Pediat, New York, NY 10032 USA

Murayama, K:
 Chiba Childrens Hosp, Dept Metab, Ctr Med Genet, Midori Ku, 579-1 Heta Cho, Chiba 2660007, Japan

Wollnik, B:
 Univ Med Ctr Gottingen, Inst Human Genet, D-37073 Gottingen, Germany

Dattani, M:
 UCL, Great Ormond St Inst Child Hlth, 30 Guilford St, London WC1N 1EH, England

Heath, KE:
 Univ Autonoma Madrid, Hosp Univ La Paz, Inst Med & Mol Genet, IdiPaz, Madrid 28046, Spain

 Univ Autonoma Madrid, Hosp Univ La Paz, Skeletal Dysplasia Multidisciplinary Unit, IdiPaz, Madrid 28046, Spain

 Ctr Invest Biomed Red Enfermedades Raras, Madrid 28029, Spain

Hickson, ID:
 Univ Copenhagen, Dept Cellular & Mol Med, Ctr Chromosome Stabil & Ctr Hlth, Blegdamsvej 3B, DK-2200 Copenhagen N, Denmark

Jackson, AP:
 Univ Edinburgh, MRC Inst Genet & Mol Med, MRC Human Genet Unit, Edinburgh EH4 2XU, Midlothian, Scotland
ISSN: 00029297





AMERICAN JOURNAL OF HUMAN GENETICS
Editorial
CELL PRESS, 50 HAMPSHIRE ST, FLOOR 5, CAMBRIDGE, MA 02139 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 103 Número: 2
Páginas: 221-231
WOS Id: 000440563700005
ID de PubMed: 30057030
imagen Green Published, Hybrid Gold, Green Accepted

MÉTRICAS