A genome-wide association study identifies new loci for factor VII and implicates factor VII in ischemic stroke etiology


Por: de Vries, PS, Sabater-Lleal, M, Huffman, JE, Marten, J, Song, C, Pankratz, N, Bartz, TM, de Haan, HG, Delgado, GE, Eicher, JD, Martinez-Perez, A, Ward-Caviness, CK, Brody, JA, Chen, MH, de Maat, MPM, Franberg, M, Gill, D, Kleber, ME, Rivadeneira, F, Soria, JM, Tang, WH, Tofler, GH, Uitterlinden, AG, Vlieg, AV, Seshadri, S, Boerwinkle, E, Davies, NM, Giese, AK, Ikram, MK, Kittner, SJ, McKnight, B, Psaty, BM, Reiner, AP, Sargurupremraj, M, Taylor, KD, Fornage, M, Hamsten, A, Marz, W, Rosendaal, FR, Souto, JC, Dehghan, A, Johnson, AD, Morrison, AC, O'Donnell, CJ, Smith, NL, INVENT Consortium, MEGASTROKE Consortium Intl

Publicada: 28 feb 2019
Resumen:
Factor VII (FVII) is an important component of the coagulation cascade. Few genetic loci regulating FVII activity and/or levels have been discovered to date. We conducted a meta-analysis of 9 genome-wide association studies of plasma FVII levels (7 FVII activity and 2 FVII antigen) among 27 495 participants of European and African ancestry. Each study performed ancestry-specific association analyses. Inverse variance weighted meta-analysis was performed within each ancestry group and then combined for a trans-ancestry meta-analysis. Our primary analysis included the 7 studies that measured FVII activity, and a secondary analysis included all 9 studies. We provided functional genomic validation for newly identified significant loci by silencing candidate genes in a human liver cell line (HuH7) using small-interfering RNA and then measuring F7 messenger RNA and FVII protein expression. Lastly, we used meta-analysis results to perform Mendelian randomization analysis to estimate the causal effect of FVII activity on coronary artery disease, ischemic stroke (IS), and venous thromboembolism. We identified 2 novel (REEP3 and JAZF1-AS1) and 6 known loci associated with FVII activity, explaining 19.0% of the phenotypic variance. Adding FVII antigen data to the meta-analysis did not result in the discovery of further loci. Silencing REEP3 in HuH7 cells upregulated FVII, whereas silencing JAZF1 downregulated FVII. Mendelian randomization analyses suggest that FVII activity has a positive causal effect on the risk of IS. Variants at REEP3 and JAZF1 contribute to FVII activity by regulating F7 expression levels. FVII activity appears to contribute to the etiology of IS in the general population.

Filiaciones:
de Vries, PS:
 Univ Texas Hlth Sci Ctr Houston, Sch Publ Hlth, Dept Epidemiol Human Genet & Environm Sci, Human Genet Ctr, Houston, TX 77030 USA

 Erasmus MC, Dept Epidemiol, Rotterdam, Netherlands

Sabater-Lleal, M:
 Inst Invest Biomed St Pau, Unit Genom Complex Dis, Barcelona, Spain

 Karolinska Inst, Dept Med, Cardiovasc Med Unit, Stockholm, Sweden

 Karolinska Univ Hosp, Ctr Mol Med, Stockholm, Sweden

Huffman, JE:
 NHLBI, Populat Sci Branch, NIH, Framingham, MA USA

 Framingham Heart Dis Epidemiol Study, Framingham, MA USA

 Vet Affairs VA Boston Healthcare Syst, Ctr Populat Genom, Jamaica Plain, MA USA

Marten, J:
 Univ Edinburgh, Western Gen Hosp, MRC, Human Genet Unit,Inst Genet & Mol Genet & Pathol, Edinburgh, Midlothian, Scotland

Song, C:
 NHLBI, Populat Sci Branch, NIH, Framingham, MA USA

 Framingham Heart Dis Epidemiol Study, Framingham, MA USA

 Uppsala Univ, Dept Med Sci, Uppsala, Sweden

 Uppsala Univ, Dept Immunol Genet & Pathol, Uppsala, Sweden

Pankratz, N:
 Univ Minnesota, Sch Med, Dept Lab Med & Pathol, Minneapolis, MN 55455 USA

Bartz, TM:
 Univ Washington, Dept Biostat, Seattle, WA 98195 USA

de Haan, HG:
 Leiden Univ, Dept Clin Epidemiol, Med Ctr, Leiden, Netherlands

Delgado, GE:
 Heidelberg Univ, Med Fac Mannheim, Dept Med 5, Mannheim, Germany

Eicher, JD:
 NHLBI, Populat Sci Branch, NIH, Framingham, MA USA

 Framingham Heart Dis Epidemiol Study, Framingham, MA USA

Martinez-Perez, A:
 Inst Invest Biomed St Pau, Unit Genom Complex Dis, Barcelona, Spain

Ward-Caviness, CK:
 US EPA, Natl Hlth & Environm Effects Res Lab, Chapel Hill, NC USA

Brody, JA:
 Univ Washington, Dept Med, Seattle, WA USA

Chen, MH:
 NHLBI, Populat Sci Branch, NIH, Framingham, MA USA

 Framingham Heart Dis Epidemiol Study, Framingham, MA USA

de Maat, MPM:
 Erasmus MC, Dept Hematol, Rotterdam, Netherlands

Franberg, M:
 Karolinska Inst, Dept Med, Cardiovasc Med Unit, Stockholm, Sweden

 Karolinska Univ Hosp, Ctr Mol Med, Stockholm, Sweden

Gill, D:
 Imperial Coll London, Dept Epidemiol & Biostat, London, England

 Imperial Coll London, Dept Stroke Med, London, England

Kleber, ME:
 Heidelberg Univ, Med Fac Mannheim, Dept Med 5, Mannheim, Germany

 Friedrich Schiller Univ Jena, Inst Nutr, Mannheim, Germany

Rivadeneira, F:
 Erasmus MC, Dept Internal Med, Rotterdam, Netherlands

Soria, JM:
 Inst Invest Biomed St Pau, Unit Genom Complex Dis, Barcelona, Spain

Tang, WH:
 Univ Minnesota, Sch Publ Hlth, Div Epidemiol & Community Hlth, Minneapolis, MN USA

Tofler, GH:
 Univ Sydney, Royal North Shore Hosp, Sydney, NSW, Australia

Uitterlinden, AG:
 Erasmus MC, Dept Internal Med, Rotterdam, Netherlands

Vlieg, AV:
 Leiden Univ, Dept Clin Epidemiol, Med Ctr, Leiden, Netherlands

Seshadri, S:
 Framingham Heart Dis Epidemiol Study, Framingham, MA USA

 Boston Univ, Dept Neurol, Boston, MA 02215 USA

 Univ Texas Hlth Sci Ctr San Antonio, Glenn Biggs Inst Alzheimers & Neurodegenerat Dis, San Antonio, TX 78229 USA

Boerwinkle, E:
 Univ Texas Hlth Sci Ctr Houston, Sch Publ Hlth, Dept Epidemiol Human Genet & Environm Sci, Human Genet Ctr, Houston, TX 77030 USA

 Baylor Univ, Coll Med, Human Genome Sequencing Ctr, Houston, TX 77030 USA

Davies, NM:
 Univ Bristol, Bristol Med Sch, MRC, Integrat Epidemiol Unit, Bristol, Avon, England

Giese, AK:
 Harvard Med Sch, Massachusetts Gen Hosp, Dept Neurol, Boston, MA 02115 USA

Ikram, MK:
 Erasmus MC, Dept Epidemiol, Rotterdam, Netherlands

 Erasmus MC, Dept Neurol, Rotterdam, Netherlands

Kittner, SJ:
 Univ Maryland, Sch Med, Dept Neurol, Baltimore, MD 21201 USA

 Baltimore VA Med Ctr, Baltimore, MD USA

McKnight, B:
 Univ Washington, Dept Biostat, Seattle, WA 98195 USA

Psaty, BM:
 Univ Washington, Dept Med, Seattle, WA USA

 Univ Washington, Dept Epidemiol, 1730 Minor Ave,Suite 1360, Seattle, WA 98101 USA

 Univ Washington, Dept Hlth Serv, Seattle, WA 98195 USA

 Kaiser Permanente Washington, Kaiser Permanente Washington Res Inst, Seattle, WA USA

Reiner, AP:
 Univ Washington, Dept Epidemiol, 1730 Minor Ave,Suite 1360, Seattle, WA 98101 USA

 Kaiser Permanente Washington, Kaiser Permanente Washington Res Inst, Seattle, WA USA

 Fred Hutchinson Canc Res Ctr, 1124 Columbia St, Seattle, WA 98104 USA

Sargurupremraj, M:
 Univ Bordeaux, INSERM, U1219, Bordeaux Populat Hlth Res Ctr, Bordeaux, France

Taylor, KD:
 Harbor UCLA Med Ctr, Los Angeles BioMed Res Inst, Dept Pediat, Inst Translat Genom & Populat Sci, Torrance, CA 90509 USA

Fornage, M:
 Univ Texas Hlth Sci Ctr Houston, Sch Publ Hlth, Dept Epidemiol Human Genet & Environm Sci, Human Genet Ctr, Houston, TX 77030 USA

 Univ Texas Hlth Sci Ctr Houston, Brown Fdn, Inst Mol Med, Houston, TX 77030 USA

Hamsten, A:
 Karolinska Inst, Dept Med, Cardiovasc Med Unit, Stockholm, Sweden

 Karolinska Univ Hosp, Ctr Mol Med, Stockholm, Sweden

Marz, W:
 Heidelberg Univ, Med Fac Mannheim, Dept Med 5, Mannheim, Germany

 Synlab Holding Deutschland GmbH, Synlab Acad, Mannheim, Germany

 Med Univ Graz, Clin Inst Med & Chem Lab Diagnost, Graz, Austria

Rosendaal, FR:
 Leiden Univ, Dept Clin Epidemiol, Med Ctr, Leiden, Netherlands

 Leiden Univ, Med Ctr, Einthoven Lab Expt Vasc Med, Leiden, Netherlands

 Leiden Univ, Med Ctr, Dept Thrombosis & Hemostasis, Leiden, Netherlands

Souto, JC:
 Hosp Santa Creu & Sant Pau, Unitat Hemostasia & Trombosi, Barcelona, Spain

Dehghan, A:
 Erasmus MC, Dept Epidemiol, Rotterdam, Netherlands

 Imperial Coll London, Dept Epidemiol & Biostat, London, England

 Imperial Coll London, Sch Publ Hlth, Publ Hlth England Ctr Environm & Hlth, MRC, London, England

 Imperial Coll London, UK Dementia Res Inst, London, England

Johnson, AD:
 NHLBI, Populat Sci Branch, NIH, Framingham, MA USA

 Framingham Heart Dis Epidemiol Study, Framingham, MA USA

Morrison, AC:
 Univ Texas Hlth Sci Ctr Houston, Sch Publ Hlth, Dept Epidemiol Human Genet & Environm Sci, Human Genet Ctr, Houston, TX 77030 USA

O'Donnell, CJ:
 NHLBI, Populat Sci Branch, NIH, Framingham, MA USA

 VA Boston Healthcare Syst, Cardiol Sect, West Roxbury, MA USA

Smith, NL:
 Univ Washington, Dept Epidemiol, 1730 Minor Ave,Suite 1360, Seattle, WA 98101 USA

 Kaiser Permanente Washington, Kaiser Permanente Washington Res Inst, Seattle, WA USA

 US Dept Vet Affairs, Seattle Epidemiol Res & Informat Ctr, Off Res & Dev, Seattle, WA USA
ISSN: 00064971





BLOOD
Editorial
AMER SOC HEMATOLOGY, 2021 L ST NW, SUITE 900, WASHINGTON, DC 20036 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 133 Número: 9
Páginas: 967-977
WOS Id: 000461501400014
ID de PubMed: 30642921
imagen Green Published, Bronze

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