Autoimmune Hypercalcemia Due to Autoantibodies Against the Calcium-sensing Receptor


Por: Minambres, I, Corcoy, R, Weetman, AP, Kemp, EH

Publicada: 1 jul 2020
Resumen:
Context: Autoimmune hypocalciuric hypercalcemia (AHH) is an acquired disorder caused by the presence of blocking autoantibodies against the calcium-sensing receptor (CaSR). Few cases of this condition have been described to date in the literature. Objective: The objectives of this study were to describe 2 patients in whom the presence of AHH was suspected and to assess the patients for the presence of CaSR antibodies. Methods: CaSR antibodies were detected and characterised by immunoprecipitation assays, CaSR peptide ELISAs, and functional assays based on the calcium-stimulated accumulation of inositol-1-phosphate in a mammalian cell line expressing the CaSR. Results: Both patients presented with an acquired form of hypocalciuric hypercalcemia. Mutational analyses of CASR, GNA11, and AP2S1 for familial hypocalciuric hypercalcemia were negative. According to the presence of Hashimoto's disease in 1 patient and latent autoimmune diabetes of adulthood and thyroid autoimmunity in the other, AHH was suspected. Immunoprecipitation assays detected CaSR antibodies in both patients. Analysis of the antibody binding sites revealed 2 main epitopes at amino acids 41-69 and 114-126. Preincubation with purified CaSR antibodies against epitope 114-126 resulted in a significant decrease in inositol-1-phophate accumulation upon calcium-stimulation of mammalian cells expressing the CaSR, suggesting that the antibodies had receptor-blocking activity. Conclusions: AHH is to be suspected in patients with an acquired biochemical pattern of PTH-dependant hypocalciuric hypercalcemia, especially in those with other concomitant autoimmune diseases. Diagnosis by means of detecting CaSR antibodies may help to better characterise this probably under-reported condition.

Filiaciones:
Minambres, I:
 Hosp Santa Creu & Sant Pau, Dept Endocrinol & Nutr, Barcelona, Spain

 Univ Autonoma Barcelona, Dept Med, Barcelona, Spain

 CIBER Diabet & Enfermedades Metab CIBER, Barcelona, Spain

Corcoy, R:
 Hosp Santa Creu & Sant Pau, Dept Endocrinol & Nutr, Barcelona, Spain

 Univ Autonoma Barcelona, Dept Med, Barcelona, Spain

 CIBER Bioingn Biomat & Nanotecnol CIBER BBN, Barcelona, Spain

Weetman, AP:
 Univ Sheffield, Dept Oncol & Metab, Sheffield, S Yorkshire, England

Kemp, EH:
 Univ Sheffield, Dept Oncol & Metab, Sheffield, S Yorkshire, England
ISSN: 0021972X





JOURNAL OF CLINICAL ENDOCRINOLOGY & METABOLISM
Editorial
ENDOCRINE SOC, 2055 L ST NW, SUITE 600, WASHINGTON, DC 20036 USA, Estados Unidos America
Tipo de documento: Article
Volumen: 105 Número: 7
Páginas: 2229-2236
WOS Id: 000565922200076
ID de PubMed: 32311038
imagen Bronze, Green Accepted

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