Analysis of the influence of PTPN22 gene polymorphisms in systemic sclerosis


Por: Diaz-Gallo, L, Gourh, P, Broen, J, Simeon, C, Fonollosa, V, Ortego-Centeno, N, Agarwal, S, Vonk, MC, Coenen, M, Riemekasten, G, Hunzelmann, N, Hesselstrand, R, Tan, FK, Reveille, JD, Assassi, S, Garcia-Hernandez, FJ, Carreira, P, Camps, MT, Fernandez-Nebro, A, de la Pena, PG, Nearney, T, Hilda, D, Gonzalez-Gay, MA, Airo, P, Beretta, L, Scorza, R, Herrick, A, Worthington, J, Pros, A, Gomez-Gracia, I, Trapiella, L, Espinosa, G, Castellvi, I, Witte, T, de Keyser, F, Vanthuyne, M, Mayes, MD, Radstake, TRDJ, Arnett, FC, Martin, J, Rueda, B

Publicada: 1 mar 2011
Resumen:
Objective Two functional single nucleotide polymorphisms (SNP) in the PTPN22 gene (rs24746601 and rs33996649) have been associated with autoimmunity. The aim of this study was to investigate the role of the R263Q SNP for the first time and to re-evaluate the role of the R620W SNP in the genetic predisposition to systemic sclerosis (SSc) susceptibility and clinical phenotypes. Methods 3422 SSc patients (2020 with limited cutaneous SSc and 1208 with diffuse cutaneous SSc) and 3638 healthy controls of Caucasian ancestry from an initial case--control set of Spain and seven additional independent replication cohorts were included in our study. Both rs33996649 and rs2476601 PTPN22 polymorphisms were genotyped by TaqMan allelic discrimination assay. A meta-analysis was performed to test the overall effect of these PTPN22 polymorphisms in SSc. Results The meta-analysis revealed evidence of association of the rs2476601 T allele with SSc susceptibility (p(FDRcorrected) = 0.03 pooled, OR 1.15, 95% CI 1.03 to 1.28). In addition, the rs2476601 T allele was significantly associated with anticentromere-positive status (p(FDRcorrected) = 0.02 pooled, OR 1.22, 95% CI 1.05 to 1.42). Although the rs33996649 A allele was significantly associated with SSc in the Spanish population (p(FDRcorrected) = 0.04, OR 0.58, 95% CI 0.36 to 0.92), this association was not confirmed in the meta-analysis (p = 0.36 pooled, OR 0.89, 95% CI 0.72 to 1.1). Conclusion The study suggests that the PTPN22 R620W polymorphism influences SSc genetic susceptibility but the novel R263Q genetic variant does not. These data strengthen evidence that the R620W mutation is a common risk factor in autoimmune diseases.

Filiaciones:
Diaz-Gallo, L:
 CSIC, Inst Parasitol & Biomed Lopez Neyra, Granada 18100, Spain

Gourh, P:
 Univ Texas Hlth Sci Ctr Houston Med Sch, Dept Internal Med, Div Rheumatol & Clin Immunogenet, Houston, TX USA

Broen, J:
 Radboud Univ Nijmegen, Med Ctr, Dept Rheumatol, NL-6525 ED Nijmegen, Netherlands

Simeon, C:
 Hosp Valle De Hebron, Med Interna Serv, Barcelona, Spain

Fonollosa, V:
 Hosp Valle De Hebron, Med Interna Serv, Barcelona, Spain

Ortego-Centeno, N:
 Hosp Clin Univ, Med Interna Serv, Granada, Spain

Agarwal, S:
 Univ Texas Hlth Sci Ctr Houston Med Sch, Dept Internal Med, Div Rheumatol & Clin Immunogenet, Houston, TX USA

Vonk, MC:
 Radboud Univ Nijmegen, Med Ctr, Dept Rheumatol, NL-6525 ED Nijmegen, Netherlands

Coenen, M:
 Radboud Univ Nijmegen, Med Ctr, Dept Human Genet, NL-6525 ED Nijmegen, Netherlands

Riemekasten, G:
 Charite, Dept Rheumatol & Clin Immunol, Berlin, Germany

Hunzelmann, N:
 Univ Cologne, Dept Dermatol, D-5000 Cologne 41, Germany

Hesselstrand, R:
 Univ Lund Hosp, Dept Rheumatol, S-22185 Lund, Sweden

Tan, FK:
 Univ Texas Hlth Sci Ctr Houston Med Sch, Dept Internal Med, Div Rheumatol & Clin Immunogenet, Houston, TX USA

Reveille, JD:
 Univ Texas Hlth Sci Ctr Houston Med Sch, Dept Internal Med, Div Rheumatol & Clin Immunogenet, Houston, TX USA

Assassi, S:
 Univ Texas Hlth Sci Ctr Houston Med Sch, Dept Internal Med, Div Rheumatol & Clin Immunogenet, Houston, TX USA

Garcia-Hernandez, FJ:
 Hosp Virgen Rocio, Med Interna Serv, Seville, Spain

Carreira, P:
 Hosp 12 Octubre, Serv Reumatol, E-28041 Madrid, Spain

Camps, MT:
 Hosp Carlos Haya, Med Interna Serv, Malaga, Spain

Fernandez-Nebro, A:
 Hosp Carlos Haya, Serv Reumatol, Malaga, Spain

de la Pena, PG:
 Hosp Ramon & Cajal, Serv Reumatol, E-28034 Madrid, Spain

Nearney, T:
 Univ Texas Med Branch Galveston, Galveston, TX USA

Hilda, D:
 Univ Texas Hlth Sci Ctr San Antonio, San Antonio, TX 78229 USA

Gonzalez-Gay, MA:
 Hosp Marques de Valdecilla, Serv Reumatol, Santander, Spain

Airo, P:
 Clin Spedali Civili, Serv Reumatol & Immunol, Brescia, Italy

Beretta, L:
 Univ Milan, Referral Ctr Syst Autoimmune Dis, Milan, Italy

Scorza, R:
 Univ Milan, Referral Ctr Syst Autoimmune Dis, Milan, Italy

Herrick, A:
 Univ Manchester, Ctr Rheumat Dis, Salford Royal NHS Fdn Trust, Manchester M13 9PL, Lancs, England

Worthington, J:
 Univ Manchester, Ctr Rheumat Dis, Salford Royal NHS Fdn Trust, Manchester M13 9PL, Lancs, England

Pros, A:
 Hosp del Mar, Serv Reumatol, Barcelona, Spain

Gomez-Gracia, I:
 Hosp Reina Sofia, Serv Reumatol, Cordoba, Spain

Trapiella, L:
 Hosp Univ Cent Asturias, Med Interna Serv, Oviedo, Spain

Espinosa, G:
 Hosp Clin Barcelona, Med Interna Serv, Barcelona, Spain

Castellvi, I:
 Hosp Santa Creu & Sant Pau, Serv Reumatol, Barcelona, Spain

Witte, T:
 Hannover Med Sch, D-3000 Hannover, Germany

de Keyser, F:
 Univ Ghent, Dept Rheumatol, Ghent, Belgium

Vanthuyne, M:
 Catholic Univ Louvain, Dept Rheumatol, B-3000 Louvain, Belgium

Mayes, MD:
 Univ Texas Hlth Sci Ctr Houston Med Sch, Dept Internal Med, Div Rheumatol & Clin Immunogenet, Houston, TX USA

Radstake, TRDJ:
 Radboud Univ Nijmegen, Med Ctr, Dept Rheumatol, NL-6525 ED Nijmegen, Netherlands

Arnett, FC:
 Univ Texas Hlth Sci Ctr Houston Med Sch, Dept Internal Med, Div Rheumatol & Clin Immunogenet, Houston, TX USA

Martin, J:
 CSIC, Inst Parasitol & Biomed Lopez Neyra, Granada 18100, Spain

Rueda, B:
 CSIC, Inst Parasitol & Biomed Lopez Neyra, Granada 18100, Spain
ISSN: 00034967
Editorial
BMJ PUBLISHING GROUP, BRITISH MED ASSOC HOUSE, TAVISTOCK SQUARE, LONDON WC1H 9JR, ENGLAND, Reino Unido
Tipo de documento: Article
Volumen: 70 Número: 3
Páginas: 454-462
WOS Id: 000286927800009
ID de PubMed: 21131644
imagen Green Accepted

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