Preclinical and clinical characterization of the selective 5-HT1A receptor antagonist DU-125530 for antidepressant treatment


Por: Scorza, MC, Llado-Pelfort, L, Oller, S, Cortes, R, Puigdemont, D, Portella, MJ, Perez-Egea, R, Alvarez, E, Celada, P, Perez, V, Artigas, F

Publicada: 1 nov 2012
Resumen:
BACKGROUND AND PURPOSE The antidepressant efficacy of selective 5-HT reuptake inhibitors (SSRI) and other 5-HT-enhancing drugs is compromised by a negative feedback mechanism involving 5-HT1A autoreceptor activation by the excess 5-HT produced by these drugs in the somatodendritic region of 5-HT neurones. 5-HT1A receptor antagonists augment antidepressant-like effects in rodents by preventing this negative feedback, and the mixed beta-adrenoceptor/5-HT1A receptor antagonist pindolol improves clinical antidepressant effects by preferentially interacting with 5-HT1A autoreceptors. However, it is unclear whether 5-HT1A receptor antagonists not discriminating between pre- and post-synaptic 5-HT1A receptors would be clinically effective. EXPERIMENTAL APPROACH We characterized the pharmacological properties of the 5-HT1A receptor antagonist DU-125530 using receptor autoradiography, intracerebral microdialysis and electrophysiological recordings. Its capacity to accelerate/enhance the clinical effects of fluoxetine was assessed in a double-blind, randomized, 6 week placebo-controlled trial in 50 patients with major depression (clinicaltrials.gov identifier NCT01119430). KEY RESULTS DU-125530 showed equal (low nM) potency to displace agonist and antagonist binding to pre- and post-synaptic 5-HT1A receptors in rat and human brain. It antagonized suppression of 5-hydroxytryptaminergic activity evoked by 8-OH-DPAT and SSRIs in vivo. DU-125530 augmented SSRI-induced increases in extracellular 5-HT as effectively as in mice lacking 5-HT1A receptors, indicating a silent, maximal occupancy of pre-synaptic 5-HT1A receptors at the dose used. However, DU-125530 addition to fluoxetine did not accelerate nor augment its antidepressant effects. CONCLUSIONS AND IMPLICATIONS DU-125530 is an excellent pre- and post-synaptic 5-HT1A receptor antagonist. However, blockade of post-synaptic 5- HT1A receptors by DU-125530 cancels benefits obtained by enhancing pre-synaptic 5-hydroxytryptaminergic function.

Filiaciones:
Scorza, MC:
 IDIBAPS, Dept Neurochem & Neuropharmacol, CSIC, IIBB, Barcelona, Spain

Llado-Pelfort, L:
 IDIBAPS, Dept Neurochem & Neuropharmacol, CSIC, IIBB, Barcelona, Spain

 CIBERSAM, Ctr Invest Biomed Red Salud Mental, Madrid, Spain

Oller, S:
 Univ Autonoma Barcelona, Dept Psychiat, Hosp Santa Creu & St Pau, E-08193 Barcelona, Spain

Cortes, R:
 IDIBAPS, Dept Neurochem & Neuropharmacol, CSIC, IIBB, Barcelona, Spain

 Ctr Invest Biomed Red Enfermedades Neurodegenerat, Madrid, Spain

 Inst Invest Biomed August Pi & Sunyer IDIBAPS, Barcelona, Spain

Puigdemont, D:
 CIBERSAM, Ctr Invest Biomed Red Salud Mental, Madrid, Spain

 Univ Autonoma Barcelona, Dept Psychiat, Hosp Santa Creu & St Pau, E-08193 Barcelona, Spain

Portella, MJ:
 CIBERSAM, Ctr Invest Biomed Red Salud Mental, Madrid, Spain

 Univ Autonoma Barcelona, Dept Psychiat, Hosp Santa Creu & St Pau, E-08193 Barcelona, Spain

Perez-Egea, R:
 CIBERSAM, Ctr Invest Biomed Red Salud Mental, Madrid, Spain

 Univ Autonoma Barcelona, Dept Psychiat, Hosp Santa Creu & St Pau, E-08193 Barcelona, Spain

Alvarez, E:
 CIBERSAM, Ctr Invest Biomed Red Salud Mental, Madrid, Spain

 Univ Autonoma Barcelona, Dept Psychiat, Hosp Santa Creu & St Pau, E-08193 Barcelona, Spain

Celada, P:
 IDIBAPS, Dept Neurochem & Neuropharmacol, CSIC, IIBB, Barcelona, Spain

 CIBERSAM, Ctr Invest Biomed Red Salud Mental, Madrid, Spain

 Inst Invest Biomed August Pi & Sunyer IDIBAPS, Barcelona, Spain

Perez, V:
 CIBERSAM, Ctr Invest Biomed Red Salud Mental, Madrid, Spain

 Univ Autonoma Barcelona, Dept Psychiat, Hosp Santa Creu & St Pau, E-08193 Barcelona, Spain

Artigas, F:
 IDIBAPS, Dept Neurochem & Neuropharmacol, CSIC, IIBB, Barcelona, Spain

 CIBERSAM, Ctr Invest Biomed Red Salud Mental, Madrid, Spain

 Inst Invest Biomed August Pi & Sunyer IDIBAPS, Barcelona, Spain
ISSN: 00071188
Editorial
WILEY, 111 RIVER ST, HOBOKEN 07030-5774, NJ USA, Reino Unido
Tipo de documento: Article
Volumen: 167 Número: 5
Páginas: 1021-1034
WOS Id: 000309599400009
ID de PubMed: 22050051
imagen Green Published

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