CSF biomarker variability in the Alzheimer's Association quality control program


Por: Mattsson, N, Andreasson, U, Persson, S, Carrillo, MC, Collins, S, Chalbot, S, Cutler, N, Dufour-Rainfray, D, Fagan, AM, Heegaard, NHH, Hsiung, GYR, Hyman, B, Iqbal, K, Lachno, DR, Lleo, A, Lewczuk, P, Molinuevo, JL, Parchi, P, Regeniter, A, Rissman, R, Rosenmann, H, Sancesario, G, Schroder, J, Shaw, LM, Teunissen, CE, Trojanowski, JQ, Vanderstichele, H, Vandijck, M, Verbeek, MM, Zetterberg, H, Blennow, K, Kaser, SA

Publicada: 1 may 2013
Resumen:
Background: The cerebrospinal fluid (CSF) biomarkers amyloid beta 1-42, total tau, and phosphorylated tau are used increasingly for Alzheimer's disease (AD) research and patient management. However, there are large variations in biomarker measurements among and within laboratories. Methods: Data from the first nine rounds of the Alzheimer's Association quality control program was used to define the extent and sources of analytical variability. In each round, three CSF samples prepared at the Clinical Neurochemistry Laboratory (Molndal, Sweden) were analyzed by single-analyte enzyme-linked immunosorbent assay (ELISA), a multiplexing xMAP assay, or an immunoassay with electrochemoluminescence detection. Results: A total of 84 laboratories participated. Coefficients of variation (CVs) between laboratories were around 20% to 30%; within-run CVs, less than 5% to 10%; and longitudinal within-laboratory CVs, 5% to 19%. Interestingly, longitudinal within-laboratory CV differed between biomarkers at individual laboratories, suggesting that a component of it was assay dependent. Variability between kit lots and between laboratories both had a major influence on amyloid beta 1-42 measurements, but for total tau and phosphorylated tau, between-kit lot effects were much less than between-laboratory effects. Despite the measurement variability, the between-laboratory consistency in classification of samples (using prehoc-derived cutoffs for AD) was high (>90% in 15 of 18 samples for ELISA and in 12 of 18 samples for xMAP). Conclusions: The overall variability remains too high to allow assignment of universal biomarker cutoff values for a specific intended use. Each laboratory must ensure longitudinal stability in its measurements and use internally qualified cutoff levels. Further standardization of laboratory procedures and improvement of kit performance will likely increase the usefulness of CSF AD biomarkers for researchers and clinicians. (C) 2013 The Alzheimer's Association. All rights reserved.

Filiaciones:
Mattsson, N:
 Univ Gothenburg, Sahlgrenska Univ Hosp, Sahlgrenska Acad,Inst Neurosci & Physiol, Clin Neurochem Lab,Dept Psychiat & Neurochem, Molndal, Sweden

 Univ Calif San Francisco, San Francisco VA Med Ctr, Ctr Imaging Neurodegenerat Dis, San Francisco, CA 94143 USA

Andreasson, U:
 Univ Gothenburg, Sahlgrenska Univ Hosp, Sahlgrenska Acad,Inst Neurosci & Physiol, Clin Neurochem Lab,Dept Psychiat & Neurochem, Molndal, Sweden

Persson, S:
 Univ Gothenburg, Sahlgrenska Univ Hosp, Sahlgrenska Acad,Inst Neurosci & Physiol, Clin Neurochem Lab,Dept Psychiat & Neurochem, Molndal, Sweden

Carrillo, MC:
 Alzheinzers Assoc, Chicago, IL USA

Collins, S:
 Univ Melbourne, Dept Pathol, Florey Inst Neurosci & Mental Hlth, Melbourne, Vic, Australia

Chalbot, S:
 New York State Inst Basic Res, Staten Isl, NY USA

Cutler, N:
 Worldwide Clin Trials, Beverly Hills, CA USA

Dufour-Rainfray, D:
 CHRU Tours, Nucl Med Lab, Tours, France

 Univ Tours, Val Loire Univ, PRES Ctr, Tours, France

 Univ Tours, Val Loire Univ, PRES Ctr, UMR INSERM Imagerie & Cerveau U930, Tours, France

Fagan, AM:
 Washington Univ, St Louis, MO USA

Heegaard, NHH:
 Statens Serum Inst, Dept Clin Biochem Immunol & Genet, DK-2300 Copenhagen, Denmark

Hsiung, GYR:
 Univ British Columbia, Vancouver, BC V5Z 1M9, Canada

Hyman, B:
 MassGen Inst Neurodegenerat Dis, Charlestown, MA USA

Iqbal, K:
 New York State Inst Basic Res, Staten Isl, NY USA

Lachno, DR:
 Eli Lilly & Co, Windlesham, Surrey, England

Lleo, A:
 Hosp Santa Creu & Sant Pau, Barcelona, Spain

Lewczuk, P:
 Univ Klinikum Erlangen, Erlangen, Germany

Molinuevo, JL:
 ICN Hosp Clin & Univ, Neurol Serv, Alzheimers Dis & Other Cognit Disorders Unit, Barcelona, Spain

 Pasqual Maragall Fdn, Barcelona, Spain

Parchi, P:
 Univ Bologna, Inst Neurol Sci, Bologna, Italy

 Univ Bologna, Dept Biomed & Neuromotor Sci, Bologna, Italy

Regeniter, A:
 Univ Basel Hosp, CH-4031 Basel, Switzerland

Rissman, R:
 Univ Calif San Diego, Sch Med, La Jolla, CA 92093 USA

Rosenmann, H:
 Hadassah Hebrew Univ, Med Ctr, Jerusalem, Israel

Sancesario, G:
 Univ Roma Tor Vergata, Gen Hosp, Rome, Italy

Schroder, J:
 Univ Klinikum Heidelberg, Sekt Gerontopsychiat, Heidelberg, Germany

Shaw, LM:
 Univ Penn, Philadelphia, PA 19104 USA

Teunissen, CE:
 Vrije Univ Amsterdam Med Ctr, Dept Clin Chem, Amsterdam, Netherlands

Trojanowski, JQ:
 Univ Penn, Philadelphia, PA 19104 USA

Vanderstichele, H:
 ADx NeuroSci, Ghent, Belgium

Vandijck, M:
 Innogenet Nv, Ghent, Belgium

Verbeek, MM:
 Radboud Univ Nijmegen, Med Ctr, Donders Inst Brain, Dept Neurol, NL-6525 ED Nijmegen, Netherlands

 Radboud Univ Nijmegen, Med Ctr, Donders Inst Brain, Dept Lab Med, NL-6525 ED Nijmegen, Netherlands

Zetterberg, H:
 Univ Gothenburg, Sahlgrenska Univ Hosp, Sahlgrenska Acad,Inst Neurosci & Physiol, Clin Neurochem Lab,Dept Psychiat & Neurochem, Molndal, Sweden

 UCL, Inst Neurol, London, England

Blennow, K:
 Univ Gothenburg, Sahlgrenska Univ Hosp, Sahlgrenska Acad,Inst Neurosci & Physiol, Clin Neurochem Lab,Dept Psychiat & Neurochem, Molndal, Sweden

Kaser, SA:
 Univ Tubingen, Tubingen, Germany
ISSN: 15525260
Editorial
WILEY, 111 RIVER ST, HOBOKEN 07030-5774, NJ USA, Estados Unidos America
Tipo de documento: Article
Volumen: 9 Número: 3
Páginas: 251-261
WOS Id: 000319183400001
ID de PubMed: 23622690
imagen Green Accepted, Green Published

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